PANCE Blueprint GI and Nutrition (8%)

Biliary Disorders (PEARLS + ReelDx)

The NCCPA™ Gastroenterology and Nutrition PANCE Content Blueprint covers three topics associated with the gallbladder

Cholelithiasis vs. Cholecystitis vs. Choledocholithiasis vs. Cholangitis

Cholelithiasis Acute Cholecystitis Choledocholithiasis Cholangitis
The presence of gallstones in the gallbladder

A precursor to cholecystitis

Inflammation of the gallbladder usually due to gallstones Presence of gallstones in the common bile duct Infection/inflammation of the bile ducts
Formation of gallstones due to cholesterol or bilirubin Blockage of cystic duct by gallstones Migration of gallstones from the gallbladder to the common bile duct Blockage of bile ducts, often due to gallstones, leading to bacterial infection
Often asymptomatic; biliary colic Severe RUQ pain, low-grade fever, nausea, vomiting, positive Murphy’s sign RUQ pain, jaundice, dark urine, pale stools Charcot's triad: RUQ pain, fever, jaundice

Reynold's pentad: add hypotension and altered mental status

Ultrasound Ultrasound, HIDA scan (gold standard) ERCP, MRCP, ultrasound ERCP, MRCP, blood cultures
Asymptomatic: observation; symptomatic: cholecystectomy Antibiotics, cholecystectomy ERCP to remove stones, followed by cholecystectomy Antibiotics, ERCP to drain the bile duct
Biliary colic, acute cholecystitis Gallbladder gangrene, perforation Pancreatitis, cholangitis Sepsis, liver abscess, recurrent cholangitis
Acute and chronic cholecystitis
Patient will present as → a 40-year-old female presents with a 24-hour history of constant, severe right upper quadrant abdominal pain. She describes the pain as sharp and radiating to her back, worsening after meals. She also reports a low-grade fever and nausea. Her past medical history includes multiple episodes of similar but less severe pain. On examination, she has a fever of 38.2°C (100.8°F), and her right upper quadrant is notably tender with a positive Murphy’s sign. Laboratory tests show elevated white blood cell count and mild elevation in liver enzymes. An abdominal ultrasound reveals a thickened gallbladder wall and gallstones, consistent with acute cholecystitis. She is admitted for intravenous antibiotics and surgical consultation for cholecystectomy.

Acute cholecystitis is persistent cystic duct obstruction by a gallstone causing gallbladder wall inflammation, presenting with RUQ pain lasting >6 hours, fever, and a positive Murphy's sign (inspiratory arrest with RUQ palpation).

  • Distinguished from biliary colic by fever and leukocytosis
  • Calculous cholecystitis (~90%): cystic duct obstruction → bile stasis → inflammation → possible secondary infection (E. coli, Klebsiella, Enterococcus); obstruction is the primary driver
  • Acalculous cholecystitis (~10%): no stones; occurs in critically ill patients (ICU, trauma, burns, TPN, sepsis); due to bile stasis + ischemia; higher mortality
  • Chronic cholecystitis: recurrent inflammation → fibrosis and wall thickening; may develop Rokitansky-Aschoff sinuses; porcelain gallbladder (calcification) has low malignancy risk unless associated with mucosal abnormalities
  • RUQ pain, constant (not colicky), >6 hours, worse with movement or inspiration
  • Fever/chills, nausea and vomiting
  • + Murphy's sign: inspiratory arrest with RUQ palpation; highly suggestive of cholecystitis
  • Leukocytosis; mild ↑ ALP, AST/ALT, bilirubin (marked elevation → think choledocholithiasis)
  • Complications: empyema, gangrenous cholecystitis (ischemia/necrosis), perforation, pericholecystic abscess, Mirizzi syndrome, gallstone ileus

DX: RUQ ultrasound: first-line — gallstones, wall thickening (>4 mm), pericholecystic fluid, sonographic Murphy's sign

  • HIDA scan (cholescintigraphy) — most sensitive test; non-visualization of the gallbladder = cystic duct obstruction
  • CT abdomen: evaluate complications or acalculous disease

TX: Laparoscopic cholecystectomy — definitive treatment; perform early (within 72 hours)

  • NPO, IV fluids, analgesia (NSAIDs or opioids), antiemetics
  • IV antibiotics: gram-negative + anaerobe coverage (piperacillin-tazobactam, ampicillin-sulbactam; ceftriaxone + metronidazole for moderate disease)
  • Percutaneous cholecystostomy: for unstable or poor surgical candidates (bridge to surgery)
  • Acalculous cholecystitis: cholecystostomy + treat underlying illness
Cholangitis
Patient will present as → a 58-year-old male with acute onset of abdominal pain associated with fever and shaking chills. The patient is hypotensive and febrile with a temperature of 102.2 ° F. Although he is confused and disoriented, he complains of right upper quadrant pain during palpation of the abdomen. His sclerae are icteric, and the skin is jaundiced

Ascending cholangitis is a life-threatening infection of the biliary tree caused by biliary obstruction (most commonly choledocholithiasis) that allows gut bacteria to ascend, producing Charcot's triad (RUQ pain + fever/chills + jaundice) and, in severe cases, Reynolds' pentad (adds hypotension + altered mental status). Urgent ERCP with biliary decompression is the definitive treatment.

  • Pathophysiology: common bile duct obstruction (choledocholithiasis = stones in the CBD is the most common cause of obstruction; also stricture, malignancy, parasites, post-ERCP) → bile stasis → bacterial overgrowth → ascending infection → bacteremia/sepsis
  • Most common organisms: E. coli (most common), Klebsiella, Enterococcus, Bacteroides
  • Charcot's triad: RUQ pain + fever/chills + jaundice (classic but not always present)
  • Reynolds' pentad: Charcot's triad + hypotension + altered mental status → severe (suppurative) cholangitis with sepsis
  • Symptoms: RUQ or epigastric pain, fever with rigors, jaundice
  • Severe disease: hypotension, confusion (septic shock)
  • Labs: leukocytosis, ↑ conjugated bilirubin, ↑ alkaline phosphatase, mild ↑ AST/ALT; blood cultures often positive
  • Ultrasound: dilated CBD (>7 mm; >10 mm post-cholecystectomy), may show stones

DX: Clinical diagnosis (Charcot/Reynolds) + imaging evidence of obstruction

  • RUQ ultrasound: first-line to detect biliary dilation and stones
  • MRCP: noninvasive, highly sensitive for choledocholithiasis if ultrasound is inconclusive
  • ERCP — diagnostic and therapeutic test of choice; allows biliary decompression, sphincterotomy, stone extraction, and stenting

TX: Immediate management: IV fluids, NPO, analgesia, obtain blood cultures

  • IV antibiotics: broad-spectrum gram-negative + anaerobe coverage (piperacillin-tazobactam, or ceftriaxone + metronidazole; severe cases may require carbapenem)
  • Urgent ERCP with biliary decompression
  • Timing: within 24 hours for most patients; emergent (within hours) if septic shock/Reynolds' pentad
  • If ERCP unavailable/unsuccessful: percutaneous transhepatic biliary drainage (PTBD) or surgical decompression
  • Elective laparoscopic cholecystectomy after recovery to prevent recurrence
Cholelithiasis
ReelDx Virtual Rounds (acute cholelithiasis)
Patient will present as → a 43-year-old woman who comes to the emergency department with a 12-hour history of right upper quadrant (RUQ) abdominal pain. The pain is severe now but waxes and wanes and is associated with nausea and some episodes of vomiting. The pain sometimes radiates through to the back. She feels warm but has not checked her temperature. There is no diarrhea. Her last bowel movement was 1 day ago and was normal. The patient has no similar history in the past. On examination, the patient is an obese young woman in some discomfort. Her vital signs reveal a temperature of 100 ° F and pulse of 102 beats/ minute. Her blood pressure is 130/70 mmHg, and her respirations are 18 breaths/minute. There is no scleral icterus. The chest is clear, and the cardiovascular examination is normal. Abdominal examination reveals marked upper abdominal tenderness with guarding, especially in the RUQ. On palpation of the RUQ of the abdomen when the patient is asked to take a deep breath, there is a marked increase in pain. The bowel sounds are present but seem slightly sluggish. The patient has no drug allergies and is not taking any medications at present.

Cholelithiasis (stones in the gallbladder) is the formation of gallstones (usually cholesterol stones, ~80% in Western countries) due to bile supersaturation and stasis.

  • Stones in the gallbladder; pain occurs from gallbladder contraction against a transiently obstructed cystic duct
  • Pathophysiology: cholesterol supersaturation → crystal formation → stones; pigment stones from ↑ bilirubin (hemolysis, cirrhosis)
  • A precursor cholecystitis (inflammation of the gallbladder)
  • Often asymptomatic (~80%); symptomatic disease = episodic RUQ pain after fatty meals lasting <6 hours (biliary colic), no fever or leukocytosis
  • Radiation to the right shoulder (referred pain via the phrenic nerve, C3–C5)
  • Radiation to the right subscapular region (Boas sign)—via thoracic nerves (T6–T9)
  • Risk factors = “4 F’s” (female, fat, forty, fertile) + rapid weight loss, pregnancy, Native American ethnicity, hemolysis (pigment stones)
  • Key differentiator: cholelithiasis = pain only vs cholecystitis = pain + fever + leukocytosis + +Murphy sign

DX: RUQ ultrasound (first-line and best initial test) showing echogenic stones with posterior acoustic shadowing; no wall thickening or pericholecystic fluid

  • Labs typically normal (no leukocytosis, normal LFTs)
  • Complications: acute cholecystitis, choledocholithiasis (↑ bilirubin/ALP), pancreatitis (↑ lipase)

TX: No intervention if asymptomatic

  • Symptomatic (biliary colic): Laparoscopic cholecystectomy — definitive treatment
  • Low-fat diet while awaiting surgery
  • Ursodeoxycholic acid: for poor surgical candidates only; slow, high recurrence
GI and Nutrition Content Blueprint Cram Session (Prev Lesson)
(Next Lesson) Brian Wallace PA-C Podcast: Diseases of the Gallbladder and Liver
Back to PANCE Blueprint GI and Nutrition (8%)

NCCPA™ CONTENT BLUEPRINT

Have you tried the NEW Smarty PANCE QBANK? It's FREE with EVERY membership purchase 😀!

X